In Epilepsy, BAD Is Not Really Bad
نویسندگان
چکیده
In this issue of Neuron, Giménez-Cassina et al. (2012) show that fuel utilization by neuronal mitochondria, controlled by the Bcl-2 family member BAD, defines response to seizures. Control of K(ATP) channels by mitochondrial metabolism might be a target for antiepileptic therapies.
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ورودعنوان ژورنال:
- Neuron
دوره 74 شماره
صفحات -
تاریخ انتشار 2012